Preliminary experiments suggest conolidine may possibly inhibit specific ion channels, minimizing neuronal excitability and limiting soreness signals. This mechanism is particularly suitable in neuropathic pain, in which abnormal signaling leads to persistent discomfort. In addition, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to discomfort perceptio... https://mattu047ycd6.wikigop.com/user