Preliminary reports suggest conolidine may inhibit certain ion channels, minimizing neuronal excitability and restricting soreness alerts. This system is especially related in neuropathic soreness, where abnormal signaling results in persistent pain. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational docking ex... https://gener470wto9.bloginder.com/profile